by
Cláudia Figueiredo-Silva, Zinpro, USA
Zinc (Zn) was identified as an essential human micronutrient in 1961. Zn
deficiency symptoms include growth retardation, immune insufficiency, chronic
inflammation, skin abnormalities, impairment of wound healing, persistent
diarrhea, loss of appetite and brain dysfunctions, among others, in both humans
and animals.
In addition, Zn is a structural constituent of ∼750 Zn-finger transcription factors enabling
gene transcription and is a catalytic component of approximately 2,000 enzymes,
encompassing all six classes (hydrolase, transferase, oxido-reductase, ligase,
lyase, and isomerase). Hence, Zn is essential for cellular processes, including
growth and development, as well as DNA synthesis and RNA transcription.
Although information on Zn functions in aquatic species is limited, the basic
metabolic functions of trace minerals (TMs) and particularly Zn are recognised
to be similar across species (NRC, 2011). Zn is known to exert beneficial
effects beyond growth performance, such as bone and nervous system development,
improvements in reproduction, reductions in oxidative stress and increased
resistance to disease in fish and shrimp.
A common cause of Zn deficiency is malnutrition. Illness and/or infection and
high phytate-containing diets reduce Zn bioavailability by inhibiting uptake.
The replacement of fish meal (FM) in aquaculture diets may significantly reduce
the Zn content while increasing levels of anti-nutritional factors, e.g. phytic
acid. Phytic acid may bind TMs and make
them less available for absorption, with direct effects on performance. As
supplements, mineral amino acid (AA) complexes have an absorption advantage
over inorganic minerals, because they are minimally antagonised by dietary
components like phytic acid and use AA-transporters instead of common metal ion
transporters (Gao et al. 2014; Sauer et al. 2017).
Read more, HERE.
The Aquaculturists
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